SNIDigital Innovations in Learning

SI Digital, Innovations and Learning, a new video journal interactive with discussion.

Now, offering this program and all of its programs on podcasts on Apple, Amazon, and Spotify,

in association with SI, Surgical Neurology, International and Internet Journal, with the MCF students, et cetera, in chief, SI Digital is pleased to present another in its investigative series

by Dr. Russell Blalock, under the title of Dr. Blalock reports, and this is on the metabolic basis of cancer, Part 3,

and he's talking about the viral cause of cancer Dr. Blyak, CEO of Theoretical Neuroscience Research. He's the associate editor-in-chief of the neuro-inflammation section of surgical neurology,

international, and SNI digital in his own both boards. He's a board certified clinical nutritionist. He's the creator and editor of the Blalock Wellness Report. He's out there in multiple books,

scientific papers. He's way ahead of his time. He's a health commentator, radio on TV, and in the epic times These are some of his books, which are available at amazoncom, on natural solutions

for the liver cures, natural strategies for cancer patients, which is a subject of today's talk, his prescriptions for natural health, excitotoxins, which is going to be the disease of the 21st

century. And he wrote with Dr. Ausman, a co-author to book on the China virus, What is the Truth? All of these are at amazoncom

He publishes a monthly newsletter. It's from Wellness Report at Newsmaxcom. And here, two of the topics. One is cancer is actually a metabolic disease. And what natural treatments are that

prevent and cure deadly colon cancer and other cancers? Well, we're happy again to be with Russell Blalock that today, and Russell is gonna present the third in a series of three

discussions that we have on starting out with the first topic that we published on SNI digital, I'll show you that now.

And here we go. And this is the homepage of SNI digital, and you see the first discussion we recorded was cancer is a metabolic disease. And we'll just go into that for just a minute, I'll stop

sharing here but basically the point there with it. that Russell was making, is that we've all grown up with the idea that cancer was a genetic disorder. And for years, people have studied cancers

and what is the

genetic changes and so forth. But it turned out that a scientist by the name of Dr. Seifried did an experiment that changed everybody's thinking and it's the foundation of what Russell's gonna

present is Dr. Seifried who had a cancer cell in a normal cell and he took the cancer cell nucleus out of the cancer cell and he transplanted it into a normal cell and nothing happened. You tell me

if I'm wrong. Yeah, once he transferred, it was just a normal cell. He had a normal cell. And then what he did is he took

a normal cell,

and he put it in a cancer cell cytoplasm. The nucleus was taken out and he put

a normal cell in there and that with the cancer cell cytoplasm and those cells grow into tumors.

Which led to the fat, well, led to the conclusion that it's really not a genetic disease because you would have expected that with all the different tumors that are formed that there would be some

final common pathway that would come out and that hasn't been happening because when they analyzed all the genetics from the tumor cells, there didn't appear to be one basic pathway in which

everything went. And so what that meant is we had to look for some other mechanisms and the mechanisms as Dr. Seifried was pointing out, or mechanisms in which the cytoplasm was transformed. into

producing cancer cells. Is that correct? That's right. And the cytoplasm changes the metabolic change, primarily. And so that was the first lecture. In other words, basically everybody had

brought up with the idea that cancer was a genetic disease and the answer is it's not a genetic disease, it's a metabolic disease And the second talk that we haven't posted yet was based on an

interview that Russell and I did, which described all the different agents that can lead to cancer coupled with when they essentially get in, for instance, if it's radiation, the radiation enters

the cell. Now, it may not alter the genetic makeup at all, Well, what it does do is produces

an activation that we'll talk about in this talk, which is a latent virus. It activates the virus and its genome to essentially direct the cancer and the metabolism cell, the cell, and its

metabolism to form a cancer cell. So the second video was on all the things that can initiate it. And correct me if I'm wrong, we had microwave radiation, which you get with your cell phone. You

get radiation, if you go and you get that with therapy, you get chemotherapy, you can get fluoride and water, which is toxic or aluminum, which can produce toxic damage to the nervous system.

And I'm sure I've left some things out, but there's a whole, or trauma, is another thing that happens. And all of these things then kick off immuno-excited or toxicity, which is another concept,

or a russell is introduced, which becomes a final common pathway to essentially,

Initially, it's a protective reaction with immunology, but then after a while, with continued insults, it becomes damaging to the cell, and you need the immunoexcited toxicity to translate the

toxic product of its trauma or toxic, those two together, and you need to produce a cancer cell, and what

Russell's going to talk about in this talk is something that is not commonly known, and that is, there has to be a viral genome in the cancer cell, and he's going to talk about how common it is,

it can be up to a hundred percent of the cells, or hundred percent in one particular virus, he'll tell you a bar about that, and that's transformed and activated and can produce a cancer cell. So

that's topic one. Cancer is a metabolic disease, cancer too. Many things can cause it tied together with immuno-excited toxicity. And number three, which he's gonna focus on today, is on how

viruses become central to forming cancer. Is that a fair summary? That's a great summary. Okay. So you wanna take it from there. And

you want me to put up the diagram that we looked at?

In just a minute Okay. You wanna start facing it? Okay. To start the discussion out. If you take a normal cell and you infect it with cytomegal bars on one of the other latent viruses, it

suddenly changes its metabolism to look exactly like we'd see in cancer cell. It's dependent on glycolysis. Normally the metabolism in the cell is a three-tier thing, glycolysis, and then Peruvate

is produced. That enters the outer membrane of the mitochondria, starts another system, system two, which is a Krebs cycle. And then that goes and produces electrons that pass through the

electron transport system, which is a third tier. And that produces altogether about 36

ATP's. Well, we find in cancer that the last two, particularly the third one is dysfunctional. And so the cancer cell is dependent on glycolysis. Okay. And I think that's kind of what your

diagram you see me today is

demonstrated. It gets to the glycolysis stage, and rather than going into a Krebs cycle and electron transport system, it produces lactic acid, which is why the cancer is acidic.

So the metabolism is driving it. It's primary fuel, which was thought forever, was glucose. And then the diagram that we show is

different. Glutamine becomes a primary fuel. And so if you look at all cancers, the primary fuel is a glutamine, and the second fuel is glucose So what you're doing is if you're treating cancers

of metabolic disease, you're starving the cancer cells to death. Normal cells don't use these systems. And so they can continue to thrive, but the cancer cell cannot. Yeah, for instance, other

cells can use ketones. Most cancer cells cannot use ketones.

So if you look at the diagram that we see up there now, We see in the red type is what happens in a cancer cell? You get inhibition of the things in the cell that normally inhibit it from becoming a

cancer cell.

So at each stage, you're seeing a change. It inhibits the P53, P21, RAS, and PTN, which normally suppresses cancer and even will kill the cell rather than let it become a cancer Well, the

cancer inhibits that. So number one, the P53 is not working anymore and neither are the other inhibitors. And so that drives glycolysis. And then you see calcium release. Well, glutamate is

acting through its receptor, allows calcium to enter the cell and calcium is a powerful driver of cancer, of cash or growth, invasion, and spread. So you can see the glutamate. In part, it's

doing that way. The glutamine is converted by an enzyme into glutamate. This infectious virus, this latent virus, is actually

driving the production of the enzyme that converts glutamine to glutamate. It's actually through the receptor, mainly to increase calcium, and that drives the cancer cell. And then you see on the

left-hand side of the diagram, the immune suppression from STAT3. There's normally three different types of STAT. Normally, STAT1 is what you use to inhibit cancer, inhibit bacteria and viruses.

STAT3 does the opposite.

STAT3 actually suppresses immunity and allows the cancer cell as well as the oncogenic virus to proliferate.

And so that's another big part of it is immune suppression. And

then the third part is cell proliferation. So you've got these things that protect the cell silenced, and now the cancer virus is stimulating the AKT and MTOR, which is a cell signaling apparatus

which actually stimulates the growth of the cell. And we see a number of things that were normal, but kept in low concentrations so that the cell can't reproduce unless it's needed. But now because

it's at a high rate, particularly the AKT and the

STAT3 and the change in the metabolism of the cell, now it's becoming immortal cell. It cannot kill itself. It cannot protect the cell from becoming cancer.

And so that's the basic process there. Okay, showing as you see on the left-hand side is growth factor simulation, which goes through P-I3K-AKT, which stimulates cell proliferation. And it does

it by increasing the amount of glucose that enters the cell. There are certain tanks transporting glucose into the cell like glute one and three And so they are pouring glucose into the cell, and it

goes through this top diagram here, which is glycolysis, to produce some ATP, and some chemicals that are needed by the virus to make more viruses. But right here, it

peruvate kinase, that M2, the peruvate, is where glycolysis ends. And normally, in the presence of oxygen, That Perubate will enter. they might have contrary. But since that's not working

right, the probate is converted to lactate. Let's see over here. It's changed to lactate as acidotic. And so the cell has changed its metabolism. It's now highly dependent on glucose. It cannot

use other things. Until you get to the over on the right-hand side, it says glutamine. And the glutamine is converted by an enzyme with the glutamate, glutaminease. And then it can end Krebs

cycle at this cycle here where it says alpha-ketoglutarate. Then it can go in a circle and it can use these substrates to make different components of the cancer cell

But it's bypassed some of the primary

sites of entry into the Krebs cycle.

And we see that sea mike, which is a oncogene, normally stimulates this breakdown of glutamine into glutamate called glutamylysis. And the cell at that point is dependent on glutamine, primarily,

not glucose. And it has to have glutamine. Glutamine's a very common amino acidicide in almost all foods, particularly meats, and the blood level is fairly high of glutamine. So if you can cut

off or reduce the amount of glutamine in the energy cell, and you can reduce the glucose, the cell starts again. And so the cancer cell will die. Well, it won't affect normal cells. Normal cells

can bypass these things and continue the metabolism normally So it's only the cancer cell is dependent on that stage one. And good me, see, normal cells aren't dependent on that.

So that's terrific. So what we're looking at here is a normal, what could be, it was a normal cell, the normal cell metabolism. And what happens is, as the

as these toxic agents are basically affect the cell, it activates the virus. And what normally happens in a cell is this is glucose, which you, the food you eat makes glucose, it gets into this

normal glycolysis pathway, and then it goes into the normal mitochondria, and the mitochondria passes through this for various sets of changes and it produces energy. And that's how there are 37

trillion cells

But what's happening is there are changes that are going on every day.

And they're stimulating the cell, maybe, and here's the stimulating with this on toxin, or radiation, whatever it is, or chemotherapy. And what that does is it converts the cell by using the

viral genome. It converts the cell because what's happening is the genome is directing the metabolism just to stop at pyruvate, it then uses glutamine, glutamine, and then it directs it down

another pathway to make more cells, more tumor cells. And here's lipid biosynthesis, nucleotides, that's what you need for DNA. So essentially, it becomes a factory for producing tumor cells.

Is that right? That's right. That's right, it's producing all the components that it needs to make a cancer cell And the reason that the virus has become important

before, we thought, well, maybe this happened if it was a genetically changed, but you'd have to expect that everything would be going through this same pattern of behavior. And that's just,

they were doing that, but it wasn't because of these very genetic changes. There wasn't a single pathway. This becomes a single pathway to convert a normal cell to a tumor cell. Is that right?

Yeah. The cypher looked at cancers and he found some cancers had no genetic change He looked at others and only some of the cells had genetic changes and they were different. Well, as

pointed out, if you're going to have a cancer, the oncogenes should affect the same thing in all the cells. But he found out it was different in all the cells. And what we see is because the

oncogenic virus produces over 200 proteins and each one of these proteins has a different function. and they can do everything we see with cancer. If you show that table one from my article, it

shows the different proteins. Let me get to that in a second here. I'm sorry Oh

I've got to get to your paper here.

Here's your paper

So, you're talking about, you can pick it up here in a second. Here you go. Let me make it a little bit. That's it. Yeah. You can shrink that down. Yeah. Make it bigger here so you can see it.

I think I just passed it by. Here you go. That's what you want to see, right? That's it. Oh, you can see it. Okay. See, each one of these things in red is a protein produced by the virus,

and the virus is producing hundreds of protein. And what they found is that with the cytomegal virus, for instance, is the virus itself is mutated and is slowly reproducing itself. Takes a very

long time to reduce any more cytomegal virus, but it's producing hundreds of protein. Now, if you look on the left at the top, you see immune suppression Well, all these proteins listed there on

the left in red, they cause immune suppression.

Then in cancer cells you see genomic instability. Well, you look at three proteins for producing that from the cytomegal virus and Then you look down here pus 28 and red Look at all it does it

activates in look in six which makes cancer deadly It activates CP one which is a chemo kind that attracts macrophages to the cancer with the the job that is supposed to kill the cancer Well, that's

called the M1 type macrophage Well, the protein switches it to M2

Which does just the opposite it's paralyzed the immunity and we call these Tam cells Tumor-associated macrophages. They're inhibitory the inhibit the immune system And so they allow the cancer cell

to grow like wow And then you see it also increases BEGF, which is a vascular endothelial growth factor, and it produces angiogenesis, more development of lycro vessels. That's a very early change

in cancer. It increases invasion and metastasis. And then you see the third thing, it activates NF-Cappa-B activation. Well, NF-Cappa-B is activated in the nucleus from the cytoplasm It

drastically enhances inflammation by increasing all the inflammatory cytokines in COX-2 and all these things are increased by NF-Cappa-B. NF-Cappa-B is activated in all tumors constantly. And then

this fourth one, you see dysregulated cellular energetics. So that one protein can produce the Warburg effect in the cell. And then the last one prolonged STAT-3 activation. So if you look at

cancer cells, instead of STAT1, which kills cancer cells, is STAT3,

which paralyzes immunity and stimulates glycolysis and causes immune suppression so that the cancer grows real fast. That's just from a single protein produced by the cytomegal virus itself When you

look down here, you've got

PUL122 and 123 and 97. They inhibit the suicide genes. Normally, your cell has suicide genes in it that if it looks like it's going to become a cancer, it has two minutes of damage to it, it

kills the cell, so it won't convert to cancer. Well, these proteins inhibit that, so it can't do it And we find if P53 is negative in a cell, it doesn't work. Actually, it increases this

glycolysis and it starts all the changes we see in a cancer cell. Then below that, you see four different proteins that inhibit apoptosis, that is the killing of the cancer cell. So it inhibits it,

so it can't kill itself.

And then on the right, you see activation of PI3K, AKT Well, those are cell signaling programs that cause cell proliferation and immortality of

the cell. So it'll just continue to proliferate. The higher the activity of this enzyme, PI3K, AKT,

the faster the proliferation. And finally, you see one protein cause activation of telomeres Well, in a cell, each strand of DNA has telomeres

the cap on shoelace. So your shoelace won unravel. Well, that's what keeps the DNA from unraveling. And each time a free radical strikes that telomere, it takes the piece of it all. Until it's

all gone, then the DNA unravels and the cell dies. Well, there's an enzyme called tenomerase, which actually reveals it so that the cell becomes immortal. Well, that protein from the side of

megavirus activates this tenomerase and makes it work even better. So you can see virtually everything you see on a cancer cell that has to do with its survival and its proliferation and its invasion

can be done by the proteins secreted by a side of megavirus. And this is just a handful of proteins. It's producing almost 200 different proteins.

And what you're, this is terrific So what you're saying. then is the

cytomeic, cytomeic. Make the whole virus. Right, is

has the genetic makeup to produce either producer direct production of these various proteins in the cell, which essentially convert the cell from a normal cell to a cancer cell And instead of the

cell having normal mechanisms from the immune system to kill it, it blocks those. It has, the cell has various things. If it goes awry to kill itself, which is a suicide gene, it stops at, so

that means the cell can't kill itself. It's gonna go on forever. And then it's gonna secrete proteins that are gonna make new blood vessels and further enhance inflammation. And then direct the

metabolism by the Warburg effect, it's gonna steal a glucose out, not going through the mitochondria, directly, it's gonna steal that out to make all these other substances like lipids and things

that you need to make, new cells and membranes and so forth. And

I think that's in, it stops the cell from, that's inhibits apoptosis and so forth, and it prolongs its life by activating the telomeres. So this

is the virus, the sonomacallic virus, that is basically containing the information that is causing this cell to turn into a tumor cell. Is that right? Yeah, and one of the mysteries they always

had, well, if this isn't happening, what is converting all these systems in the cell? What is doing it? Well, they couldn't find anything until they found these proteins secreted by the virus.

Well, everything we see in a cancer cell can be explained by these proteins. it can induce it. And so now we know the connection between a latent virus or an oncogene, an ocogenetic virus

that is offering the

cell to produce all the changes we see in a cancer cell. Now, the other mystery was, when you look at a cancer cell, some of the tumors, they say, Well, we didn't find any virus in. And others,

we only found some of the cells were infected with the virus. And they're so, if most of the tumor cells did not have virus in it, how are they being converted? Well, now we know. We found that,

in fact, the

cytomegal virus, for instance, infects primarily cancer stem cell, or stem cell. And it converts them to cancer stem cell then all of his daughter cells are cancer cells. You don't have the virus

in it, it has the protein produced by the virus in it. And that's what's doing it, it's the mass number of proteins it's producing, it's changing all the other cells to cancer, which you won't

find a virus in it, you'll find the virus in the stem cell.

And we find that older cells tend also to have the cyanomega virus in it and other latent viruses and we call these synolytic cells or synagenic

cells. So as your body gets older, you have these scattered all over your bodies, these cells, these cells are highly inflammatory. Well what's the common denominator for all cancer,

inflammation? Inflammation activates these latent viruses If you produce inflammation and an experimental animal infected with the latent virus, until the infection. Our inflammation takes hold.

They remain asleep. Once your immune system is suppressed, they wake back up. And then they can do all these things that we're talking about. They can change

all the cytoplasmic metabolism, the production of

angiogenesis, suppression of tumor associated genes and proliferation. So every aspect of cancer we're seeing with these infected

cells or the cells that come from infected cells.

So now the audience is gonna be saying, Well, if you really are, you're saying that all the cells in the body are certainly a significant number of them have to be infected with a virus, but the

virus may be latent, it's not doing anything. Is that in other words, for all this to happen, you need the virus there, in most cases, right? where you don't need it in all cells. You just

need it in a very small percentage of the cell. Okay. For instance, like most of the

stem cells in your body are located in the hemaquatic system. They're just sitting there asleep.

If you have a cytomegal virus infection, now they become infected. But they're still latent inside the stem cell If they're activated, the virus now converts the stem cell to a cancer stem cell.

Now it may become different kinds of cancer. If it's in the brain, in the brain, they've been tricklers area, and the sub-ventricular zone is where most of the stem cells are located. They're

just sitting quiet until there's an injury to the brain and they're needed. What if they're infected with a latent a megabirus or another virus.

They're producing these proteins that now convert it to a tumor So you get a glioblastoma name and they look at glioblastoma Almost a hundred percent of them contains that omega box And one of the

other bars So if you look at the cell rest of the cells in the body where look at the heart we look at the liver We look at the kidneys and lungs and so forth There have to be what you're saying is

stem

cells everywhere and those are the preferential still cells That are activator that have virus in it, but then the virus is activated in the stem cell. Is that correct? That's right So so does if

you're if somebody has a cytomegolic Virus infection would you expect the virus to be in all the cells in the body or does the virus just infect the stem cells? Just the stem cell. Okay before they

got all cell But they since learned didn't have to be an old cell Okay. But all the cells have to be affected by the proteins, those fused stem cells produce.

So, whenever the target organizes, there's stem cells which have been infected by a virus and it may be latent. And what's happened is it's activated by some toxin or trauma or something, and that

activates

the immuno-excitotoxicity system, which turns on, it converts the latent virus to an active virus, is that correct? That's right. In other words, you have a process that if it's

immuno-excitotoxicity that's occurring for a long period, that's producing inflammation throughout your body and excitotoxicity, both of which produce high levels of free radicals and liver

peroxidation products which activate latent viruses. Okay

So, so then. So that's then the fundamental concept of how we get a cancer cell. Now, what other kind of viruses can do this? I mean, you said cytomegalic virus, are there other viruses that

can be act similarly? Yeah, there's a number of latent viruses like herpes simplex one and two, herpes simplex virus six and seven, and the Epstein-Barr virus has the 40 bars. So there's a

papilloma virus, a number of them, can act in this manner in which they're making cancers, but they're doing it primarily through their protein they produce, not by infecting the cell. And we

find that actually viral reproduction is suppressed whilst making the protein

So this would be for different - different types of virus infection or what? Well, there are different types of viruses. For instance, some people found that glioblastoma has infection with the

herpes simplex, like one. Others have found a high percentage of cytomegal virus in glioblastoma. One thing about activating the

herpes simplex virus, I mean, activating cytomegal virus is that it suppresses that stem cell from becoming a neuron, it preferentially becomes an astrocyte. Well, what are most tumors in the

brain? They're not

from neurons,

they're from astrocytes. Now, is the herpes virus, we didn't talk about this before, but does herpes virus cause some different kind of pathways to be changed? Or is it causing it to change in

the same way This is side of my galaxy virus where it seemed to be the same mechanism.

Okay, so it takes it over from the virus. It becomes a fundamentally similar mechanism, which produces

a tumor in each organ or at each different location in front. So now, what else do we didn't talk about in terms of the viruses,

how do we treat this? What do we do about this? How do we, if the viruses are latent, how can you take care of them? Well, one thing that we know about latent viruses, they're only activated if

your immune system is weak. And so, when we look at aging, what are the people who get cancers when they're older? Well, gradually, in a lot of aged people, the immune system sorts faltering,

it gets weak. That activates these hidden viruses, these latent viruses. And so, then they cause cancer. They could be there for the whole life of the person. They never had any knowledge that

they even were infected. It produced no problem until these cells got older and the immune system got weaker. Now we've had the COVID virus and you've written about this. And with the COVID

infection and then we're just some recent papers that said what you had said some time ago, how the vaccination suppressed the immune system And I think you mentioned before that it takes, how many

vaccinations does it take to wipe out your immune system? Well, by the third vaccine, they're getting significant suppression of the immune system. After that, it can become tremendous. And

that's when you start seeing these turbo cancers. So that then was gonna lead you into that. So we're seeing a rise in cancers in young people. Could this be one of the mechanisms by which that

happens? it very well be because a lot of these viruses like the cytomegal virus, even the herpes virus, are inherent in their past from the mother through the years to the baby. And so the baby

may carry the latent virus until the virus is activated. Now we know if a cytomegal virus, for instance, is activated in a child, fully activated can kill the child. Experiments have shown that

if you take a cytomegal virus and you change its latency, so it's producing these tumor genetic proteins, it will make the external granular layer of the cerebellum ticking. Well, one of the most

common childhood tumors is the

medulloblastoma. The number one virus found in medulloblastoma, which is almost found always, the cytomegal virus. So here we have direct evidence that the cytomegal virus can cause tickening and

overgrowth of the tissue. that are known to cause a major oblastoma.

Do we have a direct connection now?

We're beginning to find the same thing in prostate cancers, colon cancer, breast cancers.

As we are sophistication and detection increases, we're finding a large number of those cancers have the viruses in them. For instance, we find a megabirus in breast cancer when you look at

prostate cancer, almost all of the prostate cancers have cytomegalovirus. When you look at prostate hypertrophy, benign prostate hypertrophy, there's no cytomegalovirus. And

that's the difference. Okay, so now if we're looking for ways to treat it, let me get back to this slide here because this is, I think you're gonna want us to explain that And we'll go to here.

We'll go back to here. And this is the normal cell with a normal metabolism which goes through the mitochondria produces the energy for the cell. But what you're saying is what's happening here is

this is being diverted by the changes that are happening, perhaps in the mutated virus, but the changes that come along with the virus to essentially stop this and hijack this pathway going down

here to make all kinds of things that are needed to make new cells. Is that right? That's right. And then some goes off here into lactate, which you, we find is in acidic environment. Now, you

talked about this before. So one of the ways you said that, and here's glutamine and you talked about glutamine can come into the cell and then take over part of the mitochondria and can begin to be

utilized also in this new construction of cells,

at least as energy for this new cell formation. So what you're saying is one of the ways to treat this is to cut off the glucose and to cut off the glutamine into the cell and essentially give them

ketones which the cell can work on but it won't grow as a tumor cell, is that right? That's right, the vast majority of tumor cells cannot use ketones to produce energy or new cells but normal

cells, you can use them just fine. Doesn't have any problem using ketone. And

so one of the treatments for your suggesting for cancer is to essentially starve the cell and give them ketones. Can an individual survive on ketones? Yeah, the normal cells can survive just fine,

cancer can I can't answer being starved to death. Okay.

eliminate glutamine, it's an essential amino acid, but you can reduce it. And it's things like EGCG, which comes from green and white tea, that will suppress glutamine. And so it drastically

lowers it and has a very powerful anti-cancer effect. In the glucose, you can fast and you can go on a ketogenic diet and reduce your intake of glucose and you can start to starve the cancer cells.

It makes them weaker. So then you use other flavonoids. It'll destroy the cell. It'll destroy the cancer cell because it comes weaker. You've done that by restricting glucose intake and reducing

glutamine intake. I have people use ketogenic diets to essentially give root of cancer Is there some writing about that? Oh yeah, this is quite a bit, quite a bit. In fact, some people with even

stage four cancer have been on these metabolic diets and cured themselves. It's gone for years. 10 years later, it's still gone. And as long as they stay on a moderate glucose reduction diet and a

moderate glutamine reduction diet, they do just fine And like I say, what you're doing is now you've weakened the cancer cell. And so if you use anti-cancer flavonoids like nano curcumin, nano

quercidin, resveratrol,

now they're very powerful anti-cancer agents.

Now, but what's commonly used, I talked to some people this week, they're getting a fellow, you know, he's getting radiation and chemotherapy, he's got, I think he had bladder cancer or

something. Well, what you're telling me then is the radiation is toxic, the chemotherapy is toxic. So what we're doing is treating it with agents that are producing it Yeah, one of the things

that the oncologists don't like to talk about is secondary and tertiary cancers induced by the treatment. And so there's certain chemotherapy combinations, for instance, it was high as a 30

incidence of the chemotherapy, producing a separate type cancer or even a different cancer that's totally different. So what you're using is forms of treatment, chemotherapy and radiation therapy,

which are inflammatory. Well, every stage of cancer is dependent on inflammation, every stage. So what sense does it make to use powerful inflammatory chemicals to treat a cancer? Well, what

you're doing is you're hoping you kill all the cancer cells before you kill the patient. And that's what happens to a number of patients. They're killed by the chemotherapy. They're killed by the

radiation therapy in combination. And recently they found, well, if you use a lot lower chemotherapy concentration, actually it's as good or better than using the super high doses that

they were using. Is this what you're saying generally known, Russell? It's in the literature. It's well known in the literature.

But most oncologists are trained in typical residency programs. They don't know about this They don't understand it.

the pharmaceutical companies have a tremendous influence. They're making huge amounts of money all for treating cancer with these expensive agents. I mean, people mortgage the houses, they lose

their savings to get chemotherapy and they die a few months later. And if we look at the statistics, and this is well known, is that survival with all the cancer treatment, typical cancer

treatment, the survival of a cancer, once it metastasizes, there's no more than five, it's 10 percent.

So that means 90 to 95 of cancers are gonna kill you, even though you spent enormous sums on this chemotherapy, destroyed your immune system, damaged your other cells, and used to be taught that,

well, these chemotherapy agents don't enter the brain. Well, the patients were all coming forward saying, I can't think any longer. I'm having a fall And so they call itKingmo Falls.

And so they found out recently, well, it does end in the brain. And it's producing this effect by damaging normal neurons, normal connections, because it's inflammatory. It's generating

immunoexcited toxicity, which is producing a lot of this, this whole problem.

So the treatments are turning out to be almost as bad, it's not worse than the original cancer How

long is it going to take for this message to percolate up?

It's been 30, 40 years.

And it doesn't matter, for instance, Ivermectin is one of the only agents that seems to affect that and then Ben Dazal, seems to affect the cancer stem cell. Normal chemotherapy has no effect on

cancer stem cells I can't for Stimza. In most cases,

over 90, 95, it doesn't affect the cancer stem cell. Well, what happens if you remove the tumor, but leave behind the cancer stem cell? It grows right back, but now it's even more aggressive.

And this is what you see in the cancer literature. Say when the cancer returns, it's more aggressive than the primer.

Now we know why it's more aggressive than it's growing faster, baiting more, metastasizing more than the original cancer.

So now we've come full circle here. We had a first discussion on why cancer is a metabolic disease. It's not a genetic disease. We've had a second program on the various

factors that are instigating factors that can precipitate this cancer and it's microwave radiation, it's radiation, that's standard radiations, it's chemotherapy, it's toxins in your diet, it's

toxins in the water, it's vaccines that can suppress the immune system. And then the last thing is that in the hand, what we're doing is triggering latent viruses which have either programs in them

that they can program the cell to produce various proteins that essentially shut off the normal function of the cell and make it into a cancer cell or it uses the genome from the virus to do the same

thing. So we've gone from cancer being a genetic disease to not being a genetic disease, being a metabolic disease and having as its central focus viruses which then help activate it and produce

cancer cells Is that like Cypherage has shown in his book? He said, You can get full-blown cancers. They have no genetic abnormalities. You can take a normal cell and just affect its cell

signaling without affecting the genetics and produce a cancer cell. Well, that flies in the face of the whole idea that it's a genetic disease. They all should be the affecting genetics. And so

what we find is actually most of the genetic mutation is an epic phenomenon produced by the inflammation We know if you have inflammation in the normal cell, you have genetic defects. It affects the

DNA. So they're observation that, oh, we're finding mutation. Well, are they all the same mutation? Or the literature doesn't say it is. Says that you've got a lot of mutations in the cells,

but they're different. Well, if they're producing a cancer, they should be similar.

You should be able to demonstrate that the Uncle Gene is controlling the cell signaling. of STAT3 of the NF Kappa B of PGE2. Well, you don't find that.

Rather than explain it, they just go off on a tangent and start talking about something else.

Okay. Is there anything we need to discuss where you haven't touched on? Well, what we're seeing is the modern treatment of cancer is not scientific. There's nothing scientific about it The more

we learn about the physiology, biochemistry of a cancer cell, the more we're seeing, well, the cancer treatment is antithetical to it. Why are you doing this? If all cancers are caused by

inflammation, why are you using highly inflammatory agents to produce them? If immune suppression is a major problem in cancer And you take an animal. that has a cancer in your suppressor's immune

system, the cancer grows like wildfire. Well, all your treatments are immune suppressors. They're powerful immune suppressors. What sense does that matter? And like I said, their struggle they

have is to kill the cancer before we kill the patient. Well, unfortunately, too often they kill the patient before they kill the cancer. And so they can come back and say, Well, it wasn't a

chemo Well, the chemo destroyed their immune system, and then the cancer killed them.

The cancer's everywhere. But why is it everywhere? Were you suppressed the immune system? Both with your radiation and your chemotherapy. Now, nobody can deny that. It's all through the

literature. It's well known that chemo therapy is a powerful immune suppressant. And then you have all these biochemical STAT3 the like changes, the NFTAPAB, the cocks too, the PGE2, it's not

even fresh. And yet you have other drugs, natural supplements, that do address these things, very powerfully. Well, do you use any of those? Nope.

And that's what's baffling. It's these people are dying and you're denying them treatments that experimentally and in humans we know work. But you won't do it.

Excellent. Make any sense? Excellent summary. Okay, I think we covered the subject unless you got anything else you wanted to say. No, I think that's good. But like I say, it explains why

older people get cancer more often. Sure. It explains why they're exposed to their diet and they're more likely to get cancer. It explains why people are consuming huge amounts aceto oils, like

corn oil, safflower or soy oil.

and their carcinogenic, why are the cardiologists promoting a carcinogenic oil?

It's well demonstrated in the literature, we know it increases inflammation dramatically. Why are you promoting it? It even increases heart attack and strokes. Yet you continue with this cradese

cholesterol theory and anything that's lower cholesterol is good And yet it's highly inflammatory and all the researchers and atherosclerosis say it's an inflammatory disease. Why are you giving an

inflammatory substance?

Why are you using statins which suppress immunity?

None of it makes biological sense. It doesn't make any sense whatsoever. Except it's a big profit maker for pharmaceutical companies That's the bottom wall.

It's an excellent explanation.

I thank you very much for I did a terrific job in these three series and explaining a very controversial subject in which there's a lot of misinformation. Yes, and the results are beginning to show

I've just found some new articles that's connecting cytomegal virus to prostate cancer, colon cancer, breast cancer. And they're finding this virus is uniformly found in these cancers. And the

mechanism by which the cytomegal virus is causing the cancer has been well worked out. And yet it's ignored by the oncology world and it's pure money. And it happened with the COVID vaccines 'cause

it suppressed the immune system. And as you said before, we get these terrible cancers, they rapidly grow on cancers in people and young people that we've never seen before. And

these cancers that are produced by it

any treatment. Of course, they haven't tried iromectin and bender's old, the traditional treatments, they don't respond and these patients die very soon, very rapidly.

Okay, well, another controversy presented, I just did a terrific job. All right, thank you very much. We appreciate it Russell. These are the key references that he has. Take some screenshots

These are the first set of references for this talk

These are the second set of references for this talk

These are the third set of references. Take a screenshot

In this slide, Dr. Blalock explains Dr. Seifried's experiments

and the references listed when a cancer nucleus is implanted into a normal cell

This is Dr. Blalock's explanation of Dr. Seifried's experiments on cancer. The reference for this is listed. When a cancer nucleus is implanted into a normal cell, specifically a cell that has

normal cytoplasm and healthy mitochondria, the resulting cell often behaves like a normal cell, despite the presence of cancerous DNA.

This experimental procedure known as nuclear transplantation

or somatic cell nuclear transfer has yielded profound insights into whether cancer is driven purely by genetic mutations or by the cell's internal environment

In more detail, he describes for the suppression of tumorogenesis, studies such as those summarized by the researchers like Thomas Seifried show that a tumor cell nucleus placed into normal

cytoplasm, especially one with healthy mitochondria, will produce normal cells

In a landmark 2004 study, the nuclei from malignant melanoma cell was translated into a nucleated mouse oocytes, and these cloned embryos developed into normal mice, demonstrating that the cancer

genome can be programmed to support normal development

Conversely, when the normal nucleus is placed into a joint, tumor cell, cytoplasm containing dysfunctional mitochondria. The resulting cells typically become tumorogenic or die, suggesting that

the cytoplasm, and particularly the mitochondria, play a critical role in

the origin of cancer. Although the cloned mice appeared to be initially healthy, they often dealt developed malignant tumors at a much faster rate than the controls. When certain cancer-related

genes were later activated, indicating that while the phenotype was suppressed, the underlying genetic risk remained.

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Cancer is a Metabolic Disease; Latent Virus Cause; Part #3 Russell Blaylock, MD

James I. Ausman, MD, MA, PhD

Russell Blaylock, MD

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Cancer is a Metabolic Disease; Latent Virus Cause; Part #3 Russell Blaylock, MD

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Russell Blaylock, MD

SNI Digital®, the Global, Interactive, Peer-reviewed, New Video Journal of Neurosurgery and Neuroscience-2024 Edition; "Innovations in Learning" for the 21st century VIDEO EDITORIAL

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